Evidence-Based Pediatric Rehabilitation

Active Kinesthetic Cueing & DMI Alignment

Engineered in close collaboration with pediatric physiotherapists to stimulate neuroplasticity and active postural reflexes without rigid immobilization.

Active Kinesthetic Cueing (BalanceImpulse)

During physical therapy, skilled clinicians rarely hold a child firmly or immobilize their joints. Instead, they apply minimal, targeted tactile pressure at key anatomical landmarks (pelvis, thoracic spine) to prompt the child's central nervous system to actively recruit stabilizing trunk musculature.

Mitti translates this clinical facilitation into ungrounded reaction wheel torque impulses with asymmetric sawtooth profiles (BalanceImpulse). Brief corrective impulses stimulate the child's proprioceptive reflex arc rather than creating mechanical dependency, enabling authentic neuroplastic motor learning.

Clinical documentary photograph of active postural equilibrium guidance
Illustrative AI visualization — not the final device

Dynamic Movement Intervention (DMI) tactile facilitation and active postural response

Neuromuscular & Sensorimotor Architecture

Pediatric Neuromuscular Pathophysiology & Sensory Deficits

Detailed clinical rationale addressing peripheral demyelinating neuropathies (CMT1B), expanded 200 ms reflex conduction delays, generalized hypotonia, and non-actuated Real2Sim baseline identification.

Peripheral NeuropathyCMT1B / MPZ

CMT1B (MPZ) Demyelination & 200 ms Reflex Latency

Charcot-Marie-Tooth Type 1B results from autosomal dominant mutations in Myelin Protein Zero (MPZ), producing severe peripheral demyelination. Nerve conduction velocities fall below 10–25 m/s (vs. normal ≥50 m/s), expanding sensorimotor reflex loops to ~200 ms.

< 10 m/s
Conduction Velocity
~200 ms
Reflex Delay

Trapezoidal DMI Pulse Shaping:

Instantaneous step impulses trigger secondary stretch-reflex recoil because delayed motor responses arrive after torque has ceased. Mitti’s 50 ms jerk-limited ramp (dT/dt ≤ 30 Nm/s), 150–300 ms sustained plateau, and 100 ms controlled ramp-down bridge this 200 ms latency window.

Source: product/sources/clinical/2026-10-cmt1b-mpz-demyelinating-neuropathy.md
Axial Core DeficitsDMI Alignment

Generalized Hypotonia & Axial Postural Collapse

Generalized muscular hypotonia presents with substantially diminished resting muscle tone, ligamentous laxity, and compromised proprioceptive feedback. When upright, children experience compensatory trunk slumping, anterior pelvic tilt, and secondary spinal curvature.

0.2 : 1.0
Distal / Proximal Ratio
≤ 1.5 Nm
Safe Sensory Cue

Assist-as-Needed Neuromuscular Trigger:

Rather than locking the skeleton in rigid plastic, minimal kinesthetic cues (≤ 1.5 Nm) trigger vestibular self-correction. Axial muscle fibers fire actively, preventing the severe muscle disuse atrophy caused by conventional passive orthotics.

Source: product/sources/00_core_system/2026-01-dynamic-movement-intervention.md
Digital Twin IdentificationMuJoCo Real2Sim

Non-Actuated LOGGING Mode & Real2Sim Identification

Prior to applying closed-loop torque, patient-specific MuJoCo physics models are identified under a strict "No Sim Cheating" discipline using passive LOGGING mode.

With motors unpowered (τ = 0.0 Nm), natural dual-IMU sway kinematics are collected to parameterize individual body segment masses, joint damping, and neural reflex delays at t = 0.

  • G1–G4 Telemetry Hierarchy: Raw IMU dynamics (G1) segment into gait events (G2) and atomic corrective primitives (G3).
  • 0.2× Habituation Downweighting (G4): Progress metrics downweight passive slumping (0.2×) while rewarding active neuromuscular recovery (1.0×).
  • Longitudinal Neuroplasticity Proof: Tracks decreasing impulse frequency over therapy weeks.
Specification: tooling/specs/simulation/clinical_pathology_modeling.md
Operational Protocol Suite

The 6 Canonical Clinical Operational Modes

Imported directly from centralized clinical data models. Each mode specifies actuation profiles, quantitative latency targets, and therapeutic neuroplasticity objectives.

Modus 1 · LoggingTier 1

Logging

Profile: Passive Kinematic Logging (0.0 Nm)
Latency: < 10 msTorque Cap: ≤ 0 Nm

Baseline postural kinematic and gait data collection without motor intervention, providing uncorrupted natural movement metrics.

Neuroplasticity: Natural compensatory motor pattern assessment.
Modus 2 · TherapyTier 2

Therapy

Profile: Active Kinesthetic Cueing (Sawtooth Pulse)
Latency: 200 msTorque Cap: ≤ 1.5 Nm

Dynamic intermittent corrective impulses with refractory rest periods that trigger the patient's own proprioceptive reflex arc rather than creating mechanical dependency.

Neuroplasticity: Active trunk stabilizer recruitment and sensory habituation prevention via sawtooth dithering.
Modus 3 · AutonomyTier 2

Autonomy

Profile: Continuous Proportional Assist-as-Needed (AAN)
Latency: < 10 msTorque Cap: ≤ 1.5 Nm

Continuous assistive balance stabilization for sustained upright posture during high-challenge functional tasks, scaling torque down as user stability improves.

Neuroplasticity: Autonomous balance equilibrium and core trunk endurance during functional play.
Modus 4 · BikeTier 3

Bike

Profile: High-Inertia Rotational Damping
Latency: < 5 msTorque Cap: ≤ 1.5 Nm

Velocity-dependent angular damping tailored for pediatric balance bike riding, suppressing high-frequency steering wobble and sudden roll collapses.

Neuroplasticity: Dynamic vestibular coordination and bilateral equilibrium control during movement.
Modus 5 · ScoliosisTier 2

Scoliosis

Profile: Differential Pelvic-Spine Unloading
Latency: < 20 msTorque Cap: ≤ 1.5 Nm

Asymmetric corrective torque modulated by differential inclination between thoracic and pelvic IMUs, discouraging lateral trunk collapse while preserving respiratory excursion.

Neuroplasticity: Active asymmetric spinal elongation and coronal trunk alignment without rigid chest restriction.
Modus 6 · Gait-RhythmTier 2

Gait-Rhythm

Profile: Proactive Rhythmic Metronome
Latency: < 10 msTorque Cap: ≤ 1.5 Nm

Alternating left-right tactile cueing synchronized to the child's walking cadence, guiding symmetric step timing and pelvic weight-shift initiation.

Neuroplasticity: Central Pattern Generator (CPG) entrainment and gait cycle cadence symmetry.

Mechatronics, Control Loops & Safety Governance

Looking for real-time 100 Hz closed-loop control diagrams, Dual-IMU APA intention gating, the 6-module hardware architecture, or the 3-stage graduated failsafe?